It is for cholesterol deposition, holesterinovite esters and neutral fat in the arteries of the intimate. Etc. are formed atherosclerotic plaques, which have three main components:
A. cellular composition - mainly smooth muscle, macrophages and T - lymphocytes
2. extracellular matrix including collagen, elastic fibers, proteoglycans and
3. intra-and extracellularly deposited lipids, cholesterol crystals
These components are found in different proportions and configurations for different lesions. Most often the surface of plaques in the aorta ateromatoznite visible fibrous layer ("top") composed of smooth muscle cells and relatively dense collagen. Below it is the necrotic core of plaque, etc.. etc. athera, containing lipids, cholesterol and cholesterol esters, detritus from dead cells and foam cells. In the periphery of the lesion is an area of neovascularisation, proliferation of small blood vessels. In advanced atherosclerotic plaques are found in calcifications. On histological preparation of the aorta is seen splitting cadovata wall between intimate and media blanks and optical processing of soluble holesterinovite crystals and lipids

Demonstration of lipids in hepatocytes is implemented using kriostatni cuts only on fresh or fixed in formalin material, but not on vlklyucheni histologically in paraffin processed tissue fragments. Thus, the material does not pass into alcoholic solutions, lipids do not dissolve and can be colored with lipoboi. The most commonly used method is coloring Sudan III, where the lipids are visualized as orange-drops in the cytoplasm of hepatocytes

Fatty liver, also known as fatty liver disease (FLD), is a reversible condition wherein large vacuoles of triglyceride fat accumulate in liver cells via the process of steatosis (i.e., abnormal retention of lipids within a cell). Despite having multiple causes, fatty liver can be considered a single disease that occurs worldwide in those with excessive alcohol intake and the obese (with or without effects of insulin resistance). The condition is also associated with other diseases that influence fat metabolism.^[1]It is difficult to distinguish alcoholic FLD from nonalcoholic FLD, and both show microvesicular and macrovesicular fatty changes at different stages.
Accumulation of fat may also be accompanied by a progressive inflammation of the liver (hepatitis), called steatohepatitis. By considering the contribution by alcohol, fatty liver may be termed alcoholic steatosis or nonalcoholic fatty liver disease (NAFLD), and the more severe forms as alcoholic steatohepatitis (part of alcoholic liver disease) and Non-alcoholic steatohepatitis (NASH).