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hotorcrazy
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It is for anemic infarction due to obturation of the thrombus or thromboembolism artery in the brain. Necrosis is kolikvatsionen type, characteristic of cells and tissues poor in protein and rich in water and lipids. When hypoxia occurs the death of brain cells, which is manifested by "softening" of brain matter, etc. encephalomalacia. Microscopy in the area of necrosis is seen stretch of pale pink colored, homogeneous, structureless granular matter with a view or as a single thread veins. Gangliyni and glial cells, and nevrofibrili not observed. On the border with preserved tissue are very large cells with bright nuclei and sometimes piknotichni swollen, vacuolated cytoplasm, which detect lipids and brown pigment / hemosiderin. The same is formed by the disintegration of erythrocytes in the hearth of necrosis and their phagocytic glial cells. These cells are ingested and dissolved lipids of myelin sheath. They are marked as lipofagi (psevdoksantomni cells) and cells derived from mikrogliyata. When staining for lipids in Sudan III kriostatni cuts on their cytoplasm are stained yellow-orange. If fatalities occur after receiving the attack (stroke), the process of necrosis ends with the formation of glial scar formation or a pseudocyst. Years later when an autopsy can be found psevdokistata attesting spent cerebral infarction.
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