4. Necrosis myocardii /=№35 Infarctus anaemicus myocardii).
This study demonstrates the preparation ischemic / anemic / coagulation necrosis in the myocardium as a result of stopping the blood flow, due mostly to the blockage of coronary artery by a clot or prolonged vasoconstriction. In anemic / white / myocardial infarction was observed well homogenized limited area of cardiomyocytes, which have lost their transverse nabrazdenost and stained more intensely than eozina. Nuclei are not clearly visible. Among the necrotic cardiomyocytes reserved visible elements of the interstitial connective tissue and blood vessels from the shadows. After 6 to 8 hours of initial clinical manifestation of myocardial necrotic fields are separate from the surrounding tissue through etc. hyperemic-hemorrhagic area represented by dilated blood vessels and bleeding, and later by inflammatory leukocytes segmented shaft
This study demonstrates the preparation ischemic / anemic / coagulation necrosis in the myocardium as a result of stopping the blood flow, due mostly to the blockage of coronary artery by a clot or prolonged vasoconstriction. In anemic / white / myocardial infarction was observed well homogenized limited area of cardiomyocytes, which have lost their transverse nabrazdenost and stained more intensely than eozina. Nuclei are not clearly visible. Among the necrotic cardiomyocytes reserved visible elements of the interstitial connective tissue and blood vessels from the shadows. After 6 to 8 hours of initial clinical manifestation of myocardial necrotic fields are separate from the surrounding tissue through etc. hyperemic-hemorrhagic area represented by dilated blood vessels and bleeding, and later by inflammatory leukocytes segmented shaft
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